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Found 32 results
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Kerkvliet, Nancy I
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W. H. Bisson
,
Koch, D. C.
,
O'Donnell, E. F.
,
Khalil, S. M.
,
Kerkvliet, N. I.
,
Tanguay, R. L.
,
Abagyan, R.
, and
Kolluri, S. Kumar
,
“
Modeling of the aryl hydrocarbon receptor (AhR) ligand binding domain and its utility in virtual ligand screening to predict new AhR ligands.
”
,
J Med Chem
, vol. 52, no. 18, pp. 5635-41, 2009.
W. H. Bisson
,
Koch, D. C.
,
O'Donnell, E. F.
,
Khalil, S. M.
,
Kerkvliet, N. I.
,
Tanguay, R. L.
,
Abagyan, R.
, and
Kolluri, S. Kumar
,
“
Modeling of the aryl hydrocarbon receptor (AhR) ligand binding domain and its utility in virtual ligand screening to predict new AhR ligands
”
,
Journal of medicinal chemistry
, vol. 52, pp. 5635–5641, 2009.
R
N. I. Kerkvliet
,
“
Recent advances in understanding the mechanisms of TCDD immunotoxicity.
”
,
Int Immunopharmacol
, vol. 2, no. 2-3, pp. 277-91, 2002.
S
A. Perkins
,
Phillips, J. Lynne
,
Kerkvliet, N. I.
,
Tanguay, R.
,
Perdew, G. H.
,
Kolluri, S.
, and
Bisson, W. H.
,
“
A structural switch between agonist and antagonist bound conformations for a ligand-optimized model of the human aryl hydrocarbon receptor ligand binding domain
”
,
Biology
, vol. 3, pp. 645–669, 2014.
A. Perkins
,
Phillips, J. Lynne
,
Kerkvliet, N. I.
,
Tanguay, R. L.
,
Perdew, G. H.
,
Kolluri, S. K.
, and
Bisson, W. H.
,
“
A Structural Switch between Agonist and Antagonist Bound Conformations for a Ligand-Optimized Model of the Human Aryl Hydrocarbon Receptor Ligand Binding Domain.
”
,
Biology (Basel)
, vol. 3, no. 4, pp. 645-69, 2014.
T
N. I. Kerkvliet
,
“
T Lymphocytes Are Direct, Aryl Hydrocarbon Receptor (AhR)-Dependent Targets of 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD): AhR Expression in Both CD4+ and CD8+ T Cells Is Necessary for Full Suppression of a Cytotoxic T Lymphocyte Response by TCDD
”
,
Toxicology and Applied Pharmacology
, vol. 185, no. 2, pp. 146 - 152, 2002.
A. K. Ehrlich
,
Pennington, J. M.
,
Bisson, W. H.
,
Kolluri, S.
, and
Kerkvliet, N. I.
,
“
TCDD, FICZ, and other high affinity AhR ligands dose-dependently determine the fate of CD4+ T cell differentiation.
”
,
Toxicol Sci
, 2018.
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